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Interactive worksheet
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Clinical contextWhy this tool matters and how to interpret it
Understand the result,
not just the number.
Post-COVID Arrhythmia Risk Model (Autonomic + ECG): Explanation and Clinical Context This model integrates autonomic markers (HRV indices) and ECG variables to estimate post-acute arrhythmic risk in individuals recovering from COVID-19. Reduced HRV (low SDNN, RMSSD) reflects sympathetic predominance and vagal withdrawal, patterns observed in post-COVID dysautonomia. QTc prolongation captures impaired repolarization reserve and drug- or inflammation-related ion channel effects.
PVC burden quantifies arrhythmic substrate and cardiomyopathy risk. Clinical features such as POTS, syncope, and pre-existing structural heart disease further increase arrhythmic vulnerability. Model Status This calculator is an evidence-informed clinical aid, not a validated risk score.
It synthesizes findings from recent post-COVID arrhythmia literature to support structured assessment and monitoring decisions.
Evidence & references6 primary sources mapped
- Source 1
Huseynov A, et al. Cardiac Arrhythmias in Post-COVID Syndrome: Prevalence and Pathology.
- Source 2
American Heart Association Scientific Statement: Cardiac Arrhythmias and Autonomic Dysfunction With/After COVID-19.
- Source 3
Menezes-Junior AS, et al. Cardiac Autonomic Function in Long COVID-19.
- Source 4
Task Force of the ESC/NASPE. Heart-rate variability standards (SDNN/RMSSD definitions and use).
- Source 5
Baman TS, et al. PVC burden and cardiomyopathy thresholds.
- Source 6
Khatib R, et al. Managing drug-induced QT prolongation. 2020.
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